By Gil Mor
Reproductive Immunology is a turning out to be sector in either immunology and reproductive sciences and helps us to raised comprehend numerous problems of being pregnant. Immunology of being pregnant is the 1st booklet to offer a whole and recent assessment of our wisdom on the topic of the position of the immune approach while pregnant and the interactions among the placenta and the maternal immune process. This new name covers intimately all of the diverse hypotheses and stories regarding the immunology of implantation and supply a realistic method for the appliance of easy reproductive immunology examine to being pregnant issues resembling preeclampsia, pre-term exertions and IUGR.
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Extra resources for Immunology of Pregnancy (Medical Intelligence Unit)
Induction of apoptosis by Toll-like receptors. Activation of the apoptotic caspase cascade by FADD in either a MyD88-dependent manner following ligation ofTLR-2, or by a MyD88-independent pathway following ligation of TLR-3. interaction of T R I P with RIP7^ Whether T L R - 3 can mediate trophoblast cell apoptosis is, as yet, unknown. ^"^'^^ Since clinical studies have shown an association between intrauterine infections and preterm labor, preeclampsia and lUGR,^'^ we hypothesize that Toll-like receptors expressed at the maternal-fetal interface may play an important role in the mechanism of pathogenesis.
MyD88-dependent and -independent signaling ofTLR-4. Activation of NF-KB by TLR-4 can occur through the classical MyD88 signaling pathway, or in a MyD88-independent manner though the recruitment of TRIE Toll-Like Receptors and Apoptosis In many cases, infection can result in apoptosis and death of infected cells and this is an important aspect of host defense. ^^ A striking finding from our studies has been the effect of peptidoglycan on first trimester trophoblast cell survival. Peptidoglycan is the major component of gram-positive bacteria and a ligand forTLR-2.
Ment Retard Dev Disabil Res Rev 2002; 8(1):3-13. 5. Romero R, Chaiworapongsa T, Espinoza J. Micronutrients and intrauterine infection, preterm birth and the fetal inflammatory response syndrome. J Nutr 2003; 133(5 Suppl 2):1668S-1673S. 6. von Dadelszen P, Magee LA, Krajden M et al. Levels of antibodies against cytomegalovirus and Chlamydophila pneumoniae are increased in early onset preeclampsia. Bjog 2003; 110(8):725-730. 7. Hsu C D , Witter PR. Urogenital infection in preeclampsia. Int J Gynaecol Obstet 1995; 49(3):271-275.