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By Tristi Muir

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Extra resources for Office Gynecology, An Issue of Obstetrics and Gynecology Clinics (The Clinics: Internal Medicine)

Sample text

Protection from HPV strains 16 and 18 has been predicted in vaccinationmodeling studies to reduce the rates of cervical cancer by 60% and reduce the risk of cervical cancer related to HPV 16 or 18 by 91% to 95% [38]. Studies appear promising that cross-protection against other high-risk HPV strains may be afforded by the HPV vaccines. Once specifics of cross-protection are clarified, these figures may be adjusted. Cost-effectiveness of vaccination is balanced against the savings in reduction of disease.

J Clin Pathol 1995;48:1. [13] Smith JS, Herrero R, Boseetti C, et al. Herpes simplex virus-2 as a human papillomavirus cofactor in the etiology of invasive cervical cancer. J Natl Cancer Inst 2002;94:1604–13. HPV – PREVENTION AND TREATMENT 215 [14] Plummer M, Herrero R, Franceschi S, et al. Smoking and cervical cancer: pooled analysis of the IARC multicentric case–control study. Cancer Causes Control 2003;14:805–14. [15] Moreno V, Bosch FX, Munoz N, et al. International agency for research on cancer.

33] Joffe H, Cohen LS, Harlow BL. Impact of oral contraceptive pill use on premenstrual mood: predictors of improvement and deterioration. Am J Obstet Gynecol 2003;189:1523–30. [34] Yonkers KA, Brown C, Pearlstein TB, et al. Efficacy of a new low-dose oral contraceptive with drospirenone in premenstrual dysphoric disorder. Obstet Gynecol 2005;106: 492–501. [35] Pearlstein TB, Bachmann GA, Zacur HA, et al. Treatment of premenstrual dysphoric disorder with a new drospirenone-containing oral contraceptive formulation.

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